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Follistatin

Also known as FS344 · Follistatin 344

Myostatin/activin antagonist — human work is largely via gene therapy, not consumer peptide vials.

Tier C-Dextreme riskadvanced
Tier C-D
Evidence
Protocol
extreme
Risk
subcut.
Route

Extreme Risk

This substance is in the extreme platform risk category due to growth-factor signaling, mitogenic potential, or complete absence of human safety data. Exercise maximum caution.

Modality Mismatch

The legitimate clinical modality for this substance (e.g., gene therapy, recombinant protein infusion) is fundamentally different from consumer 'peptide vial' products. Consumer peptide versions may not deliver the same biological activity.

Evidence visual

Follistatin evidence and risk matrix

advanced researcher fit

Evidence

Tier C-D

Risk

extreme

Regulatory

not approved

WADA

unknown

FDA

unknown

Route

subcutaneous

Mixed evidence profile. Useful for comparison, not a standalone protocol decision.

Overview

Follistatin antagonizes myostatin and activin signaling, modulating muscle growth pathways. In legitimate clinical exploration, human work includes gene transfer approaches (e.g., AAV1.CMV.huFS344 protocols) — not comparable to consumer 'peptide vial' narratives. The gap between consumer marketing and legitimate clinical modality is large. Systemic muscle-growth modulation carries theoretical off-target risks; treat as extreme risk.

Decision path

Where Follistatin fits in the research path.

Use the evidence and risk profile first, then compare vendors and run the quiz before turning this compound into a protocol decision.

1 related goal
  1. 01

    Read evidence

  2. 02

    Check risk

  3. 03

    Compare options

  4. 04

    Build plan

Peptide research path

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Research Details

Mechanism of Action

Antagonizes myostatin and activin signaling, modulating muscle growth pathways.

Administration Routes

subcutaneous

Study Dose Range

Gene therapy: vector genome dosing (not comparable to consumer peptide vials).

Expected Effects

Myostatin inhibition / muscle growth narrative. Legitimate modality is gene therapy, not RUO peptide.

Dosing Timeline

Gene therapy effects measured over months. Consumer peptide timelines are speculative.

Contraindications

No consumer-grade data.

Adverse Effects

Systemic muscle-growth modulation carries theoretical off-target risks; extreme risk.

Interaction Notes

Unknown; likely interacts with anabolic pathways and endocrine status.

Cost at a glance

No reliable cycle cost estimate yet. We need cleaner listing price and pack-size data before showing a trustworthy number.

Known Interactions

Avoid

IGF-1 LR3

Growth-factor cluster; strong clinical-only gating recommended.

Avoid

PEG-MGF

Growth-factor cluster; insufficient safety basis for consumer routing.

Frequently Compared

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CJC-1295

Tier B

Ipamorelin

Tier B-C

GHRP-2

Tier B

GHRP-6

Tier B

Information provided for educational and research reference only. Not medical advice. Not for diagnosing, treating, curing, or preventing disease. Products referenced are labeled Research Use Only (RUO) by vendors; not for human or veterinary use.

Sources and review notes

  1. Certain Bulk Drug Substances for Use in Compounding that May Present Significant Safety Risks - U.S. Food and Drug Administration - accessed 2026-05-15

    Used for FDA compounding-risk context and peptide safety flags.

  2. The Prohibited List - World Anti-Doping Agency - accessed 2026-05-15

    Used for athlete-facing WADA risk and peptide-class restrictions.

  3. Peptide therapeutics: current status and future directions - PubMed / Nature Reviews Drug Discovery - accessed 2026-05-15

    Used for broad peptide-therapeutics background and evidence framing.

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